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I was looking at the today — this is the ranked list of how the schools fared overall. You’ll recognize the big names there.
You can of the winning photojournalists.
You can in the writing categories.
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Sadly, the Hearst folks have posted only the entries from , which was brand-new last year.
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Dr. James M. Cummings, chief of the division of urology at Saint Louis University School of Medicine, said, "What is further significant is the growing body of literature linking the onset of erectile dysfunction to the symptomatic onset of coronary artery disease, thus giving clinicians a true window of opportunity to evaluate and prevent significant ischemic heart disease in a subgroup of men.
Despite considerable recent experimentation in animal models and human volunteers, information on the central pathways of erection remains cursory at best.18 It is known that androgens play a predominantly modulating role by their effect on libido and sexual behaviour. Testosterone enhances sexual interest and the frequency of sexual acts; it increases the frequency of nocturnal erections but does not effect reflexogenic or psychogenic erections.19
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Diabetes mellitus: Erectile dysfunction tends to develop 10-15 years earlier in diabetic men than among nondiabetic men. In a population study of men with type I diabetes for more than 10 years, erectile dysfunction was reported by 55% of men between the ages of 50 to 60 years. The increased risk of erectile dysfunction among men with diabetes mellitus may be due to the earlier onset and greater severity of atherosclerosis that narrows the arteries and thereby reduces the delivery of blood to the penis. When insufficient blood is delivered to the penis, it is not possible to achieve an erection.
Erectile tissue, which is contained within the corpora cavernosa above the urethra, two main arteries and several veins and nerves.
Erectile dysfunction. Two rigid cylinders have been placed into the corpora cavernosa. This type of implant has no inflation mechanism but provides adequate rigidity to the penis to allow penetration.
Flow cytometry. Single cell suspensions from spleens or tumors were stained with PE-conjugated anti–mouse CD8 (CD8-PE; BD Biosciences), allophycocyanin-conjugated anti–mouse CD4 (CD4-allophycocyanin; BD Biosciences), FITC-conjugated anti–mouse CD11c, PE-conjugated anti–mouse B7.2, cychrome (Cy)-conjugated anti–mouse MHC class II, or with allophycocyanin-conjugated anti–mouse CD11b (BD Biosciences) and PE-conjugated anti–mouse Gr-1 (CD8-PE). IL-4R expression was determined on purified CD11b+ cells with PE-conjugated anti–mouse CD124 (BD Biosciences). Isotype-matched antibodies were used as controls, and live cells were gated based on 7-amino-actinomycin D, annexin V staining. Samples were run on a flow cytometer (FACSCalibur; BD Biosciences), and the data were analyzed using FCSexpress software (v 2.0; De Novo Software).
Hypertension (high blood pressure)
Nocturnal penile tumescence (NPT): This test measures a man's erectile function while he is sleeping. Normally, a man will have five or six erections while asleep. A lack of these erections may indicate there is a problem with nerve function or circulation to the penis. The test uses two methods, the snap gauge method and the strain gauge method. The snap gauge method is performed by wrapping three plastic bands of varying strength around the penis. Erectile function is then measured based on which of the three bands breaks. The strain gauge method works by placing elastic bands around the tip and base of the penis. If the penis becomes erect during the night, the bands stretch, measuring the changes in penile circumference.
This is another test of nerve function in the shaft of the penis. This test uses electromagnetic vibration to evaluate sensitivity. A decreased perception of vibration may indicate nerve damage in the pelvic area, which can lead to impotence.
Restriction of blood flow caused by a vascular disease can occur because of a buildup of cholesterol and other substances that may block blood vessels. It also is known that blood vessels open more widely (relax) or close down to become narrower (constrict), possibly due to signals from cells called endothelial cells that are located inside the lining of these blood vessels. These signals can result in the restriction of blood flow to certain organs even without the vessels' necessarily being clogged by any material. When endothelial function is impaired, constriction that is similar to plugged blood vessels may result. This could be one explanation why ED is so often associated with vascular disease.
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Diabetes: Diabetes can cause nerve and artery damage that can make achieving an erection difficult. Between 35% and 50% of men with diabetes experience ED, according the National Institutes of Health. Some estimates are higher, stating that up to 75% of men with diabetes will experience at least some degree of ED during their lifetime and the risk increases with age.
"Erectile dysfunction is often caused by vascular disease," explained Dr. Ian Thompson, professor and chairman of the department of urology at the University of Texas Health Science Center at San Antonio. "A man could perceive decreased blood flow to the penis as being a less strong, a weaker erection, and that may actually be one of the first indicators of blood vessel disease."
Men who are dissatisfied with their penis size tend to hold that view throughout their lives
Results: Of the 2,010 men interviewed, 257 (12.8%) reported ED. The prevalence increased with age, from 2% in men aged 18-39 to 48% in those >70 years (tested for trend, p = 0.0001). A history of cardiopathy, diabetes, hypertension, neuropathy, thrombotic/hemorrhagic stroke, peripheral vascular disorders, pelvic/medullary injury and pelvic surgery/radiation all increased the risk of ED. The association of hypertension and diabetes tends to increase the risk of ED. In comparison with nondiabetic and nonhypertensive men, the odds ratio (OR) was 1.4 (95% confidence interval (CI), 0.7-3.2) for hypertensive men without diabetes, 4.6 (95% CI, 1.6-13.7) for diabetic men without hypertension and 8.1 (95% CI, 1.2-55.0) for men with diabetes and hypertension. In comparison with never smokers, the OR of ED was 1.7 (95% CI, 1.2-2.4) for current smokers and 1.6 (95% CI, 1.1-2.3) for ex-smokers and increased with duration of the habit.
The first published efficacy trial of sildenafil involved only 12 patients, but it is the only study that included objective measurements of penile rigidity.20 In the first phase of this study, penile rigidity was measured using plethysmography during visual sexual stimulation in patients taking varying doses of sildenafil or placebo. In the men who took the 50-mg dose of sildenafil, the mean duration of erection was increased by 10 minutes at the base of the penis and six minutes at the tip of the penis compared with placebo. The second phase of this study involved a diary of erectile activity in which 10 of 12 patients reported improved erectile activity while receiving sildenafil.
However, the development of complete erectile dysfunction could be predicted from the frequency of morning erections. Among men with less than one morning erection a week, the risk of developing erectile dysfunction was 2.5-fold greater than among men who had two to three morning erections per week.
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